On the herpes family, SARS-CoV-2, and the activation of chronic viral infections
❦ SARS-CoV-2 infection precipitates a molecular cascade that reactivates latent viral agents.
Infection doesn’t just pass through the body’s defenses but rather reprograms them.
It reactivates dormant pathogens and perpetuates a cycle of chronic immune activation.
Long COVID sufferers are burdened with a significantly higher prevalence of immune responses to certain DNA viruses – namely, Epstein-Barr Virus (EBV)* and Parvovirus B19 – than those in good health.
* Epstein-Barr Virus (EBV) is a common human virus that spreads primarily through saliva.
It is a member of the herpes virus family and is found all over the world.
Most people will get infected with EBV in their lifetime and will not have any symptoms.
Elevated levels of antibodies against these viruses in Long COVID patients not only signal potential viral reactivations.
It also suggests a reality where SARS-CoV-2 may be inciting a smoldering activation of chronic viral infections.
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📖 (9 Nov 2023 ~ European Heart Journal) Sequential activation of DNA viruses by the RNA virus SARS-CoV-2 in patients with long COVID syndrome ➤
© 2023 Outbreak Updates ➲
📖 (9 Nov 2023 ~ European Heart Journal) Sequential activation of DNA viruses by the RNA virus SARS-CoV-2 in patients with long COVID syndrome ➤
© 2023 European Heart Journal.







